<HashMap><database>bioimages</database><scores/><additional><omics_type>Unknown</omics_type><submitter/><full_dataset_link>https://www.ebi.ac.uk/biostudies/studies/S-BIAD1470</full_dataset_link><repository>bioimages</repository><figure_sub>Specimen</figure_sub><figure_sub>Image analysis</figure_sub><figure_sub>Study Component</figure_sub><figure_sub>organisation</figure_sub><figure_sub>Biosample</figure_sub><figure_sub>Associations</figure_sub><figure_sub>Image acquisition</figure_sub><pubmed_authors>Megan Hochstrasser</pubmed_authors><pubmed_authors>Tara Essock-Burns</pubmed_authors><pubmed_authors>David Mets</pubmed_authors><pubmed_authors>Audrey Bell</pubmed_authors><pubmed_authors>Ryan Lane</pubmed_authors><pubmed_authors>Cameron Dale MacQuarrie</pubmed_authors></additional><is_claimable>false</is_claimable><name>Data underlying the pub "Rescuing Chlamydomonas motility in mutants modeling spermatogenic failure"</name><description>We used Chlamydomonas reinhardtii motility mutants with disrupted genes homologous to human SPEF2 or DNALI1 to model spermatogenic failure disorders SPGF43 and SPGF83, respectively. We recovered aspects of wild-type motility in these mutants with a small-scale drug screen.</description><dates><release>2024-11-08T00:00:00Z</release><modification>2024-11-06T18:49:34.432Z</modification><creation>2024-11-06T18:49:34.432Z</creation></dates><accession>S-BIAD1470</accession><cross_references/></HashMap>