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null. 1, 42.
nullviji@ebi.ac.ukEMBL-EBIBIOMD0000000831Hypothalamic regulation of pituitary secretion of luteinizing hormone. II. Feedback control of gonadotropin secretion.Smith W R WRSocial, Regulations, Control, regulation, Controls, Social Control, Regulation, Social Controls., Formal Social Controls, Formal Social Controlbiochemical pathways, extent, Regulations, AW488255, hypophysis cerebri, Sectors, Public Sectors, exocrine gland fluid, Tb11, YB, exocrine gland fluid or secretion, gland, pituitary body, NetrinA, AUTSX5, Bioengineerings, number, D430049E23Rik, inflammation of pituitary gland, Copyrights, NOVH, hypophysis, CCN3, QM, Social Controls, prevention, pituitary, Hormone, FBXW4, netrin, Hp, Yb, Hek6, Biological, Cek6, Public Enterprise, prevention and control, gonadotrophin secretion, Enterprises, Formal Social Controls, Bio-Engineering, CG2706, fs(1)M104, ENSMUSG00000074119, ERP, APUDoma, Erp, Elkh, Biological Engineering, reference sample, catabolism, Luteozyman, Ebi, EBI, Public Domains, Tyrosine-protein kinase receptor EPH-2, EK6, Lutropin, Luteoziman, Bodily, SAP-2, Sap-2, IGFBP9, Public Enterprises, DmelCG4063, Social, IBP-9, preventive measures, glandula pituitaria, neuroendocrine tumour, secreted substance, Kiaa4053, hypophysitides, Secretion, Abstract, 2.7.10.1, Solute carrier family 6 member 2, bodily secretion, L10, Secretions, Etrp, CT27014, NET1, SLC6A5, biotransformation, nervous system, Tbl1, TBL1, NAT1, netA, NOVh, ICSH (Interstitial Cell Stimulating Hormone), Enterprise, NET, Net, Controlled, Bio engineerings, Luteinizing, Controlling, Elk, ELK, preventive therapy, C130099E04Rik, degradation, Formal Social Control, completeness, Neuronally-expressed EPH-related tyrosine kinase, external secretion, DmelCG2706, Interstitial Cell Stimulating Hormone, EPH tyrosine kinase 2, DXS648, SAP2, SMAP55, 9330129L11, Luteinizing hormone, neuroendocrine tumor, net, Bio engineering, neuroendocrine neoplasm, presence., engineerings, exocrine gland fluid/secretion, LH (Luteinizing Hormone), Bodily Secretion, count in organism, Social Control, Norepinephrine transporter, IGFBP-9, engineering, Public, Public Domain, Domains, EPH-like kinase 6, secretion, University, NOV, PlexA1, Domain, Data Base, Plxn1, CG4063, biodegradation, pituitary gland, prophylaxis, Engineering, Control, nov, hEK6, Controls, Interstitial Cell-Stimulating Hormone, CG18657, E-2f, mKIAA4053, E-2g, fs(1)Y[b], l(2)k16213, DmelCG18657, Sector, Bio, control, EPHT2, pituitary gland inflammation, C130088N23Rik, EG:95B7.8, 2600013D04Rik, regulation, PLXN1, Feedbacks, DXS648E, Regulation, exocrine gland secretion, netrin A, Interstitial Cell-Stimulatingbiochemical pathways, Luteinizing, Regulations, hypophysis cerebri, Controlling, preventive therapy, exocrine gland fluid, degradation, Formal Social Control, exocrine gland fluid or secretion, gland, external secretion, pituitary body, Interstitial Cell Stimulating Hormone, inflammation of pituitary gland, Luteinizing hormone, hypophysis, Social Controls, prevention, pituitary, Hormone, exocrine gland fluid/secretion, LH (Luteinizing Hormone), Bodily Secretion, gonadotrophin secretion., Hp, Social Control, secretion, prevention and control, Formal Social Controls, biodegradation, reference sample, pituitary gland, catabolism, Luteozyman, prophylaxis, Control, Lutropin, Luteoziman, Bodily, Controls, Interstitial Cell-Stimulating Hormone, Social, preventive measures, glandula pituitaria, secreted substance, hypophysitides, Secretion, bodily secretion, control, pituitary gland inflammation, Secretions, biotransformation, nervous system, regulation, Feedbacks, ICSH (Interstitial Cell Stimulating Hormone), Regulation, exocrine gland secretion, Interstitial Cell-Stimulating, ControlledfalseSmith1980 - Hypothalamic Regulation
This a model from the article:
Hypothalamic regulation of pituitary secretion of luteinizing hormone.II. Feedback control of gonadotropin secretion.
Smith WR Bull Math Biol.
(1980) 42(1): 57-78 6986927
,
Abstract:
No Abstract Available
This model was taken from the CellML repository
and automatically converted to SBML.
The original model was:
smith,1980,version02
The original CellML model was created by:
Lloyd, Catherine, May
c.lloyd@auckland.ac.nz
The University of Auckland
Auckland Bioengineering Institute
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2024-08-222024-09-022009-03-25BIOMD0000000831SBO:00001796986927C25513MODEL7898438988BIOMD0000000831GO:0032274GO:00322759913B0FMT3Q9I999Q5I3B1