<HashMap><database>biostudies-literature</database><scores/><additional><omics_type>Unknown</omics_type><volume>155(6)</volume><submitter>Brelidze TI</submitter><funding>National Cancer Institute</funding><funding>NCI NIH HHS</funding><funding>National Institute of General Medical Sciences</funding><funding>NIGMS NIH HHS</funding><journal>The Journal of general physiology</journal><pagination>e202313333</pagination><full_dataset_link>https://www.ebi.ac.uk/biostudies/studies/S-EPMC10114541</full_dataset_link><repository>biostudies-literature</repository><pubmed_title>Heteromeric wild-type/mutant potassium channel subunit composition as a major determinant of channelopathy phenotype in heterozygous patients.</pubmed_title><pmcid>PMC10114541</pmcid><funding_grant_id>R01 GM124020</funding_grant_id><funding_grant_id>R01 CA252969</funding_grant_id><funding_grant_id>R01CA252969</funding_grant_id><funding_grant_id>R01GM124020</funding_grant_id><pubmed_authors>Brelidze TI</pubmed_authors></additional><is_claimable>false</is_claimable><name>Heteromeric wild-type/mutant potassium channel subunit composition as a major determinant of channelopathy phenotype in heterozygous patients.</name><description/><dates><release>2023-01-01T00:00:00Z</release><publication>2023 Jun</publication><modification>2025-04-26T14:04:18.308Z</modification><creation>2025-04-06T14:23:08.02Z</creation></dates><accession>S-EPMC10114541</accession><cross_references><pubmed>37058331</pubmed><doi>10.1085/jgp.202313333</doi></cross_references></HashMap>