{"database":"biostudies-literature","file_versions":[],"scores":null,"additional":{"submitter":["Aslan IH"],"funding":["Stanford University Center for Innovation in Global Health","NIAID NIH HHS","Programme d'Appui Stratégique à la Recherche Scientifique","Fundação de Amparo à Pesquisa do Estado de São Paulo","National Institutes of Health","Natural Environment Research Council","National Institute of General Medical Sciences","Stanford Woods Institute for the Environment","NIGMS NIH HHS","National Science Foundation"],"pagination":["e0011836"],"full_dataset_link":["https://www.ebi.ac.uk/biostudies/studies/S-EPMC11207148"],"repository":["biostudies-literature"],"omics_type":["Unknown"],"volume":["18(6)"],"pubmed_abstract":["The geographical range of schistosomiasis is affected by the ecology of schistosome parasites and their obligate host snails, including their response to temperature. Previous models predicted schistosomiasis' thermal optimum at 21.7°C, which is not compatible with the temperature in sub-Saharan Africa (SSA) regions where schistosomiasis is hyperendemic. We performed an extensive literature search for empirical data on the effect of temperature on physiological and epidemiological parameters regulating the free-living stages of S. mansoni and S. haematobium and their obligate host snails, i.e., Biomphalaria spp. and Bulinus spp., respectively. We derived nonlinear thermal responses fitted on these data to parameterize a mechanistic, process-based model of schistosomiasis. We then re-cast t"],"journal":["PLoS neglected tropical diseases"],"pubmed_title":["Re-assessing thermal response of schistosomiasis transmission risk: Evidence for a higher thermal optimum than previously predicted."],"pmcid":["PMC11207148"],"funding_grant_id":["2019/23593-3","NE/T013710/1","R01 AI102918","DEB-2011147","T32 GM144273","R01 AI168097","T32GM144273","R35 GM133439","CEH/NSF ICER-2024383 and NSF-EEID DEB 2011179","R35GM133439, R01AI168097, R01AI102918","19/23593"],"pubmed_authors":["Diakite NR","Ozretich RW","Liu P","Sokolow SH","Pourtois JD","N'Goran EK","Monteiro AMV","Ouattara M","Pereira TA","Little DC","Athni TS","Mitchell KR","Gatto M","De Leo GA","Norman R","Mordecai EA","Chamberlin AJ","Kirk D","Mari L","Wood CL","Casagrandi R","Palasio RGS","Lwiza KM","Brierley AS","Allan F","Aslan IH","Tuan R","Yu A"],"additional_accession":[]},"is_claimable":false,"name":"Re-assessing thermal response of schistosomiasis transmission risk: Evidence for a higher thermal optimum than previously predicted.","description":"The geographical range of schistosomiasis is affected by the ecology of schistosome parasites and their obligate host snails, including their response to temperature. Previous models predicted schistosomiasis' thermal optimum at 21.7°C, which is not compatible with the temperature in sub-Saharan Africa (SSA) regions where schistosomiasis is hyperendemic. We performed an extensive literature search for empirical data on the effect of temperature on physiological and epidemiological parameters regulating the free-living stages of S. mansoni and S. haematobium and their obligate host snails, i.e., Biomphalaria spp. and Bulinus spp., respectively. We derived nonlinear thermal responses fitted on these data to parameterize a mechanistic, process-based model of schistosomiasis. We then re-cast t","dates":{"release":"2024-01-01T00:00:00Z","publication":"2024 Jun","modification":"2025-04-20T02:15:27.361Z","creation":"2025-04-20T02:15:27.361Z"},"accession":"S-EPMC11207148","cross_references":{"pubmed":["38857289"],"doi":["10.1371/journal.pntd.0011836"]}}