biostudies-literature00440Taub MNHLBINHLBI NIH HHS419-22https://www.ebi.ac.uk/biostudies/studies/S-EPMC4710796biostudies-literatureUnknown6This data article is concerned with chronic regulation of Na,K-ATPase by catecholamines. After a chronic treatment, inhibition of Na,K-ATPase activity was observed in cultures with dopamine, while a stimulation was observed in cultures treated with norepinephrine. Following a chronic incubation with guanabenz, an α adrenergic agonist, an increase in Na,K-ATPase α and β subunit mRNAs was observed. This data supports the research article entitled, "Renal proximal tubule Na, K-ATPase is controlled by CREB regulated transcriptional coactivators as well as salt inducible kinase 1" (Taub et al. 2015) [1].Data in briefData on Na,K-ATPase in primary cultures of renal proximal tubule cells treated with catecholamines.PMC47107961RO1 HL6976-01R01 HL069676Taub MCutuli F44falseData on Na,K-ATPase in primary cultures of renal proximal tubule cells treated with catecholamines.This data article is concerned with chronic regulation of Na,K-ATPase by catecholamines. After a chronic treatment, inhibition of Na,K-ATPase activity was observed in cultures with dopamine, while a stimulation was observed in cultures treated with norepinephrine. Following a chronic incubation with guanabenz, an α adrenergic agonist, an increase in Na,K-ATPase α and β subunit mRNAs was observed. This data supports the research article entitled, "Renal proximal tubule Na, K-ATPase is controlled by CREB regulated transcriptional coactivators as well as salt inducible kinase 1" (Taub et al. 2015) [1].2016-01-01T00:00:00Z2016 Mar2024-12-04T00:49:37.841Z2019-03-27T02:06:48ZS-EPMC47107962686605110.1016/j.dib.2015.12.013