<HashMap><database>biostudies-literature</database><scores/><additional><omics_type>Unknown</omics_type><volume>186(21)</volume><submitter>Ohki R</submitter><pubmed_abstract>A spontaneous mutant isolated in the presence of a high concentration of puromycin acquired a multidrug-resistant phenotype. Expression of the bmr3 gene was dramatically increased. A base substitution, T to A at the +4 position, detected in the mutant resulted in the stabilization of bmr3 mRNA.</pubmed_abstract><journal>Journal of bacteriology</journal><pagination>7450-5</pagination><full_dataset_link>https://www.ebi.ac.uk/biostudies/studies/S-EPMC523217</full_dataset_link><repository>biostudies-literature</repository><pubmed_title>Increased stability of bmr3 mRNA results in a multidrug-resistant phenotype in Bacillus subtilis.</pubmed_title><pmcid>PMC523217</pmcid><pubmed_authors>Ohki R</pubmed_authors><pubmed_authors>Tateno K</pubmed_authors></additional><is_claimable>false</is_claimable><name>Increased stability of bmr3 mRNA results in a multidrug-resistant phenotype in Bacillus subtilis.</name><description>A spontaneous mutant isolated in the presence of a high concentration of puromycin acquired a multidrug-resistant phenotype. Expression of the bmr3 gene was dramatically increased. A base substitution, T to A at the +4 position, detected in the mutant resulted in the stabilization of bmr3 mRNA.</description><dates><release>2004-01-01T00:00:00Z</release><publication>2004 Nov</publication><modification>2025-04-27T00:40:53.058Z</modification><creation>2019-03-27T01:08:15Z</creation></dates><accession>S-EPMC523217</accession><cross_references><pubmed>15489457</pubmed><doi>10.1128/JB.186.21.7450-7455.2004</doi><doi>10.1128/jb.186.21.7450-7455.2004</doi></cross_references></HashMap>