<HashMap><database>biostudies-literature</database><scores><citationCount>0</citationCount><reanalysisCount>0</reanalysisCount><viewCount>46</viewCount><searchCount>0</searchCount></scores><additional><omics_type>Unknown</omics_type><volume>6(1)</volume><submitter>Yucel O</submitter><pubmed_abstract>Many bacteria from different phylogenetic groups are able to degrade eukaryotic steroid compounds, but the underlying metabolic pathways are still not well understood. Novosphingobium sp. strain Chol11 is a steroid-degrading alphaproteobacterium. Its genome sequence reveals that it lacks several genes for steroid degradation known to exist in other model organisms.</pubmed_abstract><journal>Genome announcements</journal><pagination>e01372-17</pagination><full_dataset_link>https://www.ebi.ac.uk/biostudies/studies/S-EPMC5754482</full_dataset_link><repository>biostudies-literature</repository><pubmed_title>Genome Sequence of the Bile Salt-Degrading Bacterium Novosphingobium sp. Strain Chol11, a Model Organism for Bacterial Steroid Catabolism.</pubmed_title><pmcid>PMC5754482</pmcid><pubmed_authors>Philipp B</pubmed_authors><pubmed_authors>Wibberg D</pubmed_authors><pubmed_authors>Kalinowski J</pubmed_authors><pubmed_authors>Yucel O</pubmed_authors><view_count>46</view_count></additional><is_claimable>false</is_claimable><name>Genome Sequence of the Bile Salt-Degrading Bacterium Novosphingobium sp. Strain Chol11, a Model Organism for Bacterial Steroid Catabolism.</name><description>Many bacteria from different phylogenetic groups are able to degrade eukaryotic steroid compounds, but the underlying metabolic pathways are still not well understood. Novosphingobium sp. strain Chol11 is a steroid-degrading alphaproteobacterium. Its genome sequence reveals that it lacks several genes for steroid degradation known to exist in other model organisms.</description><dates><release>2018-01-01T00:00:00Z</release><publication>2018 Jan</publication><modification>2024-11-20T13:58:32.992Z</modification><creation>2019-03-27T03:06:23Z</creation></dates><accession>S-EPMC5754482</accession><cross_references><pubmed>29301902</pubmed><doi>10.1128/genomeA.01372-17</doi></cross_references></HashMap>