{"database":"biostudies-literature","file_versions":[],"scores":null,"additional":{"submitter":["Rehman ZU"],"funding":["National Natural Science Foundation of China","Shanghai Key Laboratory of Veterinary Biotechnology","National Key Research and Development Program of China","Biotechnology and Biological Sciences Research Council"],"pagination":["94"],"full_dataset_link":["https://www.ebi.ac.uk/biostudies/studies/S-EPMC6148804"],"repository":["biostudies-literature"],"omics_type":["Unknown"],"volume":["49(1)"],"pubmed_abstract":["Avian avulaviruses serotype 1 (abbreviated as APMV-1 for the historical name avian paramyxovirus 1) are capable of infecting a wide spectrum of avian species with variable clinical symptoms and outcomes. Ease of transmission has allowed the virus to spread worldwide with varying degrees of virulence depending upon the virus strain and host species. The emergence of new virulent genotypes from global epizootics, and the year-to-year genomic changes in low and high virulence APMV-1 imply that distinct genotypes of APMV-1 are simultaneously evolving at different geographic locations across the globe. This vast genomic diversity may be favoured by large variety of avian species susceptibility to APMV-1 infection, and by the availability of highly mobile wild birds. It has long been considered "],"journal":["Veterinary research"],"pubmed_title":["Pathobiology of Avian avulavirus 1: special focus on waterfowl."],"pmcid":["PMC6148804"],"funding_grant_id":["BB/M008681/2","2018YFD0500100","31530074","klab201702"],"pubmed_authors":["Meng C","Rehman ZU","Umar S","Ding C","Munir M","Sun Y","Mahrose KM"],"additional_accession":[]},"is_claimable":false,"name":"Pathobiology of Avian avulavirus 1: special focus on waterfowl.","description":"Avian avulaviruses serotype 1 (abbreviated as APMV-1 for the historical name avian paramyxovirus 1) are capable of infecting a wide spectrum of avian species with variable clinical symptoms and outcomes. Ease of transmission has allowed the virus to spread worldwide with varying degrees of virulence depending upon the virus strain and host species. The emergence of new virulent genotypes from global epizootics, and the year-to-year genomic changes in low and high virulence APMV-1 imply that distinct genotypes of APMV-1 are simultaneously evolving at different geographic locations across the globe. This vast genomic diversity may be favoured by large variety of avian species susceptibility to APMV-1 infection, and by the availability of highly mobile wild birds. It has long been considered ","dates":{"release":"2018-01-01T00:00:00Z","publication":"2018 Sep","modification":"2025-04-19T13:02:49.135Z","creation":"2019-03-26T23:57:00Z"},"accession":"S-EPMC6148804","cross_references":{"pubmed":["30231933"],"doi":["10.1186/s13567-018-0587-x"]}}