{"database":"biostudies-literature","file_versions":[],"scores":null,"additional":{"submitter":["Bastiaanse H"],"funding":["PFR Pipfruit Core programme","New Zealand Foundation for Science, Research and Technology"],"pagination":["159-72"],"full_dataset_link":["https://www.ebi.ac.uk/biostudies/studies/S-EPMC6638522"],"repository":["biostudies-literature"],"omics_type":["Unknown"],"volume":["17(2)"],"pubmed_abstract":["Apple scab, caused by the fungal pathogen Venturia inaequalis, is one of the most severe diseases of apple worldwide. It is the most studied plant-pathogen interaction involving a woody species using modern genetic, genomic, proteomic and bioinformatic approaches in both species. Although 'Geneva' apple was recognized long ago as a potential source of resistance to scab, this resistance has not been characterized previously. Differential interactions between various monoconidial isolates of V. inaequalis and six segregating F1 and F2 populations indicate the presence of at least five loci governing the resistance in 'Geneva'. The 17 chromosomes of apple were screened using genotyping-by-sequencing, as well as single marker mapping, to position loci controlling the V. inaequalis resistance "],"journal":["Molecular plant pathology"],"pubmed_title":["Scab resistance in 'Geneva' apple is conditioned by a resistance gene cluster with complex genetic control."],"pmcid":["PMC6638522"],"funding_grant_id":["1433","C02X0406","C06X0810"],"pubmed_authors":["Deng C","Kirk C","Gardiner SE","Bus VG","Bassett HC","Bastiaanse H","Groenworld R","Chagne D"],"additional_accession":[]},"is_claimable":false,"name":"Scab resistance in 'Geneva' apple is conditioned by a resistance gene cluster with complex genetic control.","description":"Apple scab, caused by the fungal pathogen Venturia inaequalis, is one of the most severe diseases of apple worldwide. It is the most studied plant-pathogen interaction involving a woody species using modern genetic, genomic, proteomic and bioinformatic approaches in both species. Although 'Geneva' apple was recognized long ago as a potential source of resistance to scab, this resistance has not been characterized previously. Differential interactions between various monoconidial isolates of V. inaequalis and six segregating F1 and F2 populations indicate the presence of at least five loci governing the resistance in 'Geneva'. The 17 chromosomes of apple were screened using genotyping-by-sequencing, as well as single marker mapping, to position loci controlling the V. inaequalis resistance ","dates":{"release":"2016-01-01T00:00:00Z","publication":"2016 Feb","modification":"2025-07-27T03:10:47.031Z","creation":"2025-05-18T11:59:58.789Z"},"accession":"S-EPMC6638522","cross_references":{"pubmed":["25892110"],"doi":["10.1111/mpp.12269"]}}