<HashMap><database>biostudies-literature</database><scores/><additional><omics_type>Unknown</omics_type><volume>9(4)</volume><submitter>Dong G</submitter><pubmed_abstract>&lt;h4>Background&lt;/h4>Cirrhotic patients with portal vein thrombosis (PVT) may have a high risk of hepatic decompensation and increased mortality. This study aimed to investigate if increased portal vein diameter is associated with PVT development.&lt;h4>Methods&lt;/h4>A total of 174 cirrhotic patients were enrolled between February 1 and August 31, 2017. All participants were divided into PVT (n=62) and non-PVT (n=112) groups based on the thrombus that was detected by ultrasonography and confirmed by computed tomography angiography (CTA).&lt;h4>Results&lt;/h4>The study participants, aged 54.7±10.5 years (PVT) and 55.8±11.6 years (non-PVT), were included in this analysis. The Child-Pugh score of PVT or non-PVT was 6.6±1.3 and 5.8±0.9, respectively. Hepatitis B virus (HBV) is the primary etiological agent</pubmed_abstract><journal>Annals of translational medicine</journal><pagination>289</pagination><full_dataset_link>https://www.ebi.ac.uk/biostudies/studies/S-EPMC7944309</full_dataset_link><repository>biostudies-literature</repository><pubmed_title>Increased portal vein diameter is predictive of portal vein thrombosis development in patients with liver cirrhosis.</pubmed_title><pmcid>PMC7944309</pmcid><pubmed_authors>Dong G</pubmed_authors><pubmed_authors>Ding H</pubmed_authors><pubmed_authors>Chen SY</pubmed_authors><pubmed_authors>Li F</pubmed_authors><pubmed_authors>Huang XQ</pubmed_authors><pubmed_authors>Zhu YL</pubmed_authors></additional><is_claimable>false</is_claimable><name>Increased portal vein diameter is predictive of portal vein thrombosis development in patients with liver cirrhosis.</name><description>&lt;h4>Background&lt;/h4>Cirrhotic patients with portal vein thrombosis (PVT) may have a high risk of hepatic decompensation and increased mortality. This study aimed to investigate if increased portal vein diameter is associated with PVT development.&lt;h4>Methods&lt;/h4>A total of 174 cirrhotic patients were enrolled between February 1 and August 31, 2017. All participants were divided into PVT (n=62) and non-PVT (n=112) groups based on the thrombus that was detected by ultrasonography and confirmed by computed tomography angiography (CTA).&lt;h4>Results&lt;/h4>The study participants, aged 54.7±10.5 years (PVT) and 55.8±11.6 years (non-PVT), were included in this analysis. The Child-Pugh score of PVT or non-PVT was 6.6±1.3 and 5.8±0.9, respectively. Hepatitis B virus (HBV) is the primary etiological agent</description><dates><release>2021-01-01T00:00:00Z</release><publication>2021 Feb</publication><modification>2025-04-18T15:31:20.801Z</modification><creation>2025-02-19T01:39:55.079Z</creation></dates><accession>S-EPMC7944309</accession><cross_references><pubmed>33708916</pubmed><doi>10.21037/atm-20-4912</doi></cross_references></HashMap>