<HashMap><database>biostudies-other</database><scores/><additional><submitter>Brickman TJ</submitter><funding>NIAID NIH HHS</funding><pagination>268-70</pagination><full_dataset_link>https://www.ebi.ac.uk/biostudies/studies/S-EPMC176585</full_dataset_link><abstract>We report the isolation and preliminary phenotypic characterization of manganese-resistant Bordetella bronchiseptica mutants with respect to deregulation of siderophore and iron-regulated protein expression. The fur gene of Bordetella pertussis was cloned by genetic complementation of this deregulated phenotype and confirmed as fur by nucleotide sequence analysis.</abstract><repository>biostudies-other</repository><data_source>Europe PMC</data_source><omics_type>Unknown</omics_type><volume>177(1)</volume><journal>Journal of bacteriology</journal><pmcid>PMC176585</pmcid><funding_grant_id>AI-31088</funding_grant_id><pubmed_authors>Brickman TJ</pubmed_authors><pubmed_authors>Armstrong SK</pubmed_authors></additional><is_claimable>false</is_claimable><name>Bordetella pertussis fur gene restores iron repressibility of siderophore and protein expression to deregulated Bordetella bronchiseptica mutants.</name><description>We report the isolation and preliminary phenotypic characterization of manganese-resistant Bordetella bronchiseptica mutants with respect to deregulation of siderophore and iron-regulated protein expression. The fur gene of Bordetella pertussis was cloned by genetic complementation of this deregulated phenotype and confirmed as fur by nucleotide sequence analysis.</description><dates><release>1995-01-01T00:00:00Z</release><publication>1995 Jan</publication><modification>2019-03-27T00:35:26Z</modification><creation>2019-03-27T00:35:26Z</creation></dates><accession>S-EPMC176585</accession><cross_references><gen>U11699</gen><pubmed>7798143</pubmed><doi>10.1128/jb.177.1.268-270.1995 </doi></cross_references></HashMap>