<HashMap><database>EGA</database><scores/><additional><omics_type>Genomics</omics_type><dataset_type>AB SOLiD 4 System;ABI_SOLID, AB 5500xl Genetic Analyzer;ABI_SOLID</dataset_type><full_dataset_link>https://ega-archive.org/datasets/EGAD00001002266</full_dataset_link><sample_count>14</sample_count><description>EGA dataset EGAD00001002266</description><repository>EGA</repository><title>Whole exome sequencing data of germline and two independent primary leukemias of five patients</title><name_synonyms>Vasp, VASP, Data Set., DmelCG15112, ENHANCER OF ATNSI ACTIVITY, MENA, NDPP1, l(2)02029, Enb, enb, ENA, Ena, CG15112, ENA/VASP</name_synonyms><description_synonyms>genetic susceptibilities, Malignant Neoplasm, single-organism developmental process, predisposition, Tissue., Neoplasms, postnatal development, familial, Benign Neoplasm, growth and development, genetic predisposition, Tumor, Malignant, genetic susceptibility, Client, Leukemias, development, Susceptibility, MT, Benign, Neoplasm, leukaemia NOS, Leucocythaemia, Genetic Predispositions, Leucocythaemias, average, susceptibility, Leucocythemia, primary cancer, acute lymphoblastic leukemia (ALL), Genetic, Genetic Predisposition, Malignancy, postnatal growth, Benign Neoplasms, Leucocythemias, Genetic Susceptibilities, Cancers, Predisposition, Children, predispositions, malignant tumor, genetic predispositions, Susceptibilities, Malignant Neoplasms, genetic, Neoplasias, Predispositions, Patient, malignant neoplasm, Clients, Genetic Susceptibility, inherited genetic, acute lymphoblastic leukaemia (ALL), Malignancies, constitutitional genetic, hereditary, growth, susceptibilities, Neoplasia, Cancer, Tumors</description_synonyms></additional><is_claimable>false</is_claimable><name>ena-DATASET-RUMC-28-07-2016-13:56:11:769-339 - samples</name><description>The contribution of genetic predisposing factors to the development of pediatric acute lymphoblastic leukemia (ALL), the most frequently diagnosed cancer in childhood, has not been fully elucidated. Children presenting with multiple de novo leukemias are more likely to suffer from genetic predisposition. Here, we selected five of these patients and analyzed the mutational spectrum of normal and malignant tissues.</description><dates><updated>2017-07-26 15:39:29</updated></dates><accession>EGAD00001002266</accession><cross_references><TAXONOMY>9606</TAXONOMY><EGA>EGAC00001000369</EGA><EGA>EGAS00001001889</EGA></cross_references></HashMap>