<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE14nnn/GSE14385/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by array</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE14385</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Response of bronchial epithelial cells to low doses of cigarette smoke condensate and subsequent demethylation agent</name><description>The study seeks to identify the epigenetic changes caused by exposure of to cigarette smoke condensate. To this goal human bronchial epithelial cells, BEAS-2B, were treated with 5-aza-2’deoxycitidine and trychostatin A (5AzaC/TSA) subsequent to a chronic exposure (1 month) to cigarette smoke condensate (CSC). As negative control served BEAS-2B cells that were untreated or treated with CSC/DMSO for one month without the subsequent application of 5Aza/TSA. Keywords: stress response</description><dates><publication>2009/08/01</publication></dates><accession>GSE14385</accession><cross_references><GSM>GSM359567</GSM><GSM>GSM359557</GSM><GSM>GSM359568</GSM><GSM>GSM359558</GSM><GSM>GSM359569</GSM><GSM>GSM359559</GSM><GSM>GSM359570</GSM><GSM>GSM359571</GSM><GSM>GSM359560</GSM><GSM>GSM359561</GSM><GSM>GSM359572</GSM><GSM>GSM359562</GSM><GSM>GSM359573</GSM><GSM>GSM359563</GSM><GSM>GSM359564</GSM><GSM>GSM359565</GSM><GSM>GSM359566</GSM><GPL>570</GPL><GSE>14385</GSE><taxon>Homo sapiens</taxon><PMID>[21095227]</PMID></cross_references></HashMap>