<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE181nnn/GSE181855/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Homo sapiens</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE181855</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Epigenomic and transcriptomic features induced by silencing of ZNF280C in colon cancer cells and knock-out of Zfp280c in mice [ChIP-Seq, ATAC-Seq]</name><description>Znic finger proteins play crucial roles in development and disease, including tumorigenesis. Here, we identifed ZNF280C as a novel oncogenic driver in colorectal cancer, and systematically studied the molecular mechanisms underlying ZNF280C-mediated malignant transformation and progression.</description><dates><publication>2022/05/20</publication></dates><accession>GSE181855</accession><cross_references><GSM>GSM5939228</GSM><GSM>GSM5939229</GSM><GSM>GSM5512958</GSM><GSM>GSM5512959</GSM><GSM>GSM5512956</GSM><GSM>GSM5512978</GSM><GSM>GSM5512957</GSM><GSM>GSM5512976</GSM><GSM>GSM5512977</GSM><GSM>GSM5512974</GSM><GSM>GSM5512975</GSM><GSM>GSM5512972</GSM><GSM>GSM5512973</GSM><GSM>GSM5512970</GSM><GSM>GSM5512971</GSM><GSM>GSM5939230</GSM><GSM>GSM5939231</GSM><GSM>GSM5939232</GSM><GSM>GSM5939233</GSM><GSM>GSM5512969</GSM><GSM>GSM5512967</GSM><GSM>GSM5512968</GSM><GSM>GSM5512965</GSM><GSM>GSM5512966</GSM><GSM>GSM5512963</GSM><GSM>GSM5512964</GSM><GSM>GSM5512961</GSM><GSM>GSM5512962</GSM><GSM>GSM5512960</GSM><GPL>20795</GPL><GPL>30209</GPL><SRA>SRP332045</SRA><GSE>181855</GSE><taxon>Homo sapiens</taxon><PMID>[35605119]</PMID></cross_references></HashMap>