<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE194nnn/GSE194279/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE194279</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>The KDM1A and KDM7A lysine demethylases cooperate to regulate androgen receptor function in prostate cancer cells [KDM1A &amp; KDM7A inhibitors]</name><description>KDM1A and KDM7A are Androgen Receptor coregulators. Here the KDM1A and KDM7A inhibitors, namoline and TCE-5002 were used to pharmacologically inhibit KDM1A and KDM7A in prosate cancer cells and the effect on androgen regulated gene expression and splicing determined.</description><dates><publication>2026/07/29</publication></dates><accession>GSE194279</accession><cross_references><GSM>GSM5832209</GSM><GSM>GSM5832210</GSM><GSM>GSM5832211</GSM><GSM>GSM5832200</GSM><GSM>GSM5832201</GSM><GSM>GSM5832202</GSM><GSM>GSM5832203</GSM><GSM>GSM5832204</GSM><GSM>GSM5832205</GSM><GSM>GSM5832206</GSM><GSM>GSM5832207</GSM><GSM>GSM5832208</GSM><GPL>18573</GPL><GSE>194279</GSE><taxon>Homo sapiens</taxon><PMID>[41870972]</PMID></cross_references></HashMap>