GEOapplication/xmlftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE197nnn/GSE197626/suppl/filelist.txtftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE197nnn/GSE197626/suppl/GSE197626_RAW.tarftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE197nnn/GSE197626/primaryOK200TranscriptomicsMus musculusExpression profiling by high throughput sequencinghttps://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE197626GEOGSEfalseImmune-mediated Tubule Atrophy Promotes Acute Kidney Injury to Chronic Kidney Disease TransitionIncomplete repair after acute kidney injury (AKI) is associated with progressive loss of tubular cell function and development of chronic kidney disease (CKD). Here, we compared the kidney single-cell transcriptomes from the mice subjected to either unilateral ischemia-reperfusion kidney injury with contralateral nephrectomy (IRI/CL-NX, in which tubule repair predominates) or unilateral IRI with contralateral kidney intact (U-IRI, in which fibrosis and atrophy predominates) to investigate the mechanism(s) underlying transition to CKD following AKI.2022/06/30GSE197626GSM5924060GSM5924061GSM5924062GSM5924063GSM5924064GSM5924065GSM5924054GSM5924066GSM5924055GSM5924067GSM5924056GSM5924057GSM5924058GSM592405921103197626Mus musculus[41101700][36951957][35986026]