<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE200nnn/GSE200622/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE200622</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>RNA-seq expression data from parental and SLX4IP-depleted D2.OR cells</name><description>SLX4IP mediates Hedgehog signaling through isoform-specific SUMOylation of GLI1 We sought to assess genome-wide transcriptomic and signaling changes induced by loss of SLX4IP in D2.OR breast cancer cells, which identified the Hedgehog pathways as being significantly disrupted by SLX4IP depletion</description><dates><publication>2026/04/01</publication></dates><accession>GSE200622</accession><cross_references><GSM>GSM6039219</GSM><GSM>GSM6039218</GSM><GSM>GSM6039220</GSM><GSM>GSM6039217</GSM><GSM>GSM6039216</GSM><GSM>GSM6039215</GSM><GPL>17021</GPL><GSE>200622</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>