<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE244nnn/GSE244331/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE244331</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>The increased expression of PLK1 (polo-like kinase 1) and its substrate MISP (mitotic spindle positioning) in intrahepatic cholangiocarcinoma tumor samples</name><description>Intrahepatic cholangiocarcinoma (iCCA) is a subtype of CCA and has a high mortality rate and a relatively poor prognosis. However, studies focusing on increased cell motility and loss of epithelial integrity during iCCA progression remain relatively scarce. We collected seven fresh tumor samples from four patients to perform RNA sequencing (RNA-seq) to determine the transcriptome profile of iCCA.</description><dates><publication>2026/09/10</publication></dates><accession>GSE244331</accession><cross_references><GSM>GSM7813690</GSM><GSM>GSM7813691</GSM><GSM>GSM7813685</GSM><GSM>GSM7813687</GSM><GSM>GSM7813686</GSM><GSM>GSM7813689</GSM><GSM>GSM7813688</GSM><GPL>21697</GPL><GPL>24676</GPL><GSE>244331</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>