<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE264nnn/GSE264207/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE264207</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Endothelial Parkin safeguards coronary microcirculation in myocardial ischemia/reperfusion injury</name><description>Gaining a deeper insight into the signals and molecular mechanisms that govern the function of coronary endothelial cells after MI/R could pave the way for the discovery of new preventative and therapeutic approaches. The functional role of Parkin in coronary microvascular injury in response to MI/R is unclear. Here, we investigated the upstream regulators and downstream effectors of Parkin against MI/R damage and examined the underlying mechanisms.</description><dates><publication>2026/04/12</publication></dates><accession>GSE264207</accession><cross_references><GSM>GSM8213971</GSM><GSM>GSM8213982</GSM><GSM>GSM8213983</GSM><GSM>GSM8213972</GSM><GSM>GSM8213980</GSM><GSM>GSM8213981</GSM><GSM>GSM8213970</GSM><GSM>GSM8213975</GSM><GSM>GSM8213976</GSM><GSM>GSM8213984</GSM><GSM>GSM8213973</GSM><GSM>GSM8213974</GSM><GSM>GSM8213979</GSM><GSM>GSM8213969</GSM><GSM>GSM8213977</GSM><GSM>GSM8213978</GSM><GPL>24247</GPL><GSE>264207</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>