<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE264nnn/GSE264529/</Other></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Caenorhabditis elegans</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE264529</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Gelsenicine disrupted the intestinal barrier of Caenorhabditis elegans</name><description>The treatment of gelsenicine resulted in the increase of nematode intestinal permeability. The toxicological mechanisms underlying this effect involved the disruption of intestinal barrier integrity, an imbalance between oxidative and antioxidant processes mediated by the SKN-1/DAF-16 pathway, and abnormal unfolded protein reaction.</description><dates><publication>2026/04/21</publication></dates><accession>GSE264529</accession><cross_references><GSM>GSM8219569</GSM><GSM>GSM8219568</GSM><GSM>GSM8219565</GSM><GSM>GSM8219567</GSM><GSM>GSM8219566</GSM><GSM>GSM8219570</GSM><GPL>26672</GPL><GSE>264529</GSE><taxon>Caenorhabditis elegans</taxon></cross_references></HashMap>