GEOapplication/xmlftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE26nnn/GSE26671/primaryOK2000000GenomicsMus musculusExpression profiling by arrayhttps://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE26671GEOGSE0falseExpression data of murine model of cardiac hypertrophyAn initial cellular change in the pathogenesis of heart failure is cardiomyocyte hypertrophy, characterized by increased cell size, enhanced protein synthesis and reactivation of fetal genes. In addition to mechanical stresses, several neurohumoral factors have been identified as potent hypertrophic agents, including angiotensin II, endothelin, and catecholamines. We used microarrays to study the gene expression during cardiac hypertrophy.2011/12/31GSE26671GSM656428GSM656429GSM656426GSM656427GSM656424GSM656425GSM656422GSM656423GSM656420GSM656431GSM656421GSM656430126126671Mus musculus