<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE270nnn/GSE270357/</Other></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE270357</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptome sequencing of palmitic acid-treated HepG2 cells upon intervention of curcumin or curcumin-copper complex</name><description>Globally, non-alcoholic fatty liver disease (NAFLD) poses a major risk to human health. The natural polyphenol curcumin (Cur) can act as a copper (Cu) transporter in a Cur-Cu complex for enhanced Cu cell loading, but the effects of such a complex on NAFLD remain unclear. HepG2 liver cancer cells were treated with palmitic acid (PA) to create an in-vitro NAFLD model. And the purpose of this study was to investigate the impacts of a Cur- Cu complex on this model, and its potential mechanisms and targets in the reduction of fatty-acid accumulation in HepG2 cells.</description><dates><publication>2026/06/19</publication></dates><accession>GSE270357</accession><cross_references><GSM>GSM8340250</GSM><GSM>GSM8340242</GSM><GSM>GSM8340243</GSM><GSM>GSM8340244</GSM><GSM>GSM8340245</GSM><GSM>GSM8340246</GSM><GSM>GSM8340247</GSM><GSM>GSM8340248</GSM><GSM>GSM8340249</GSM><GPL>34284</GPL><GSE>270357</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>