<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE271nnn/GSE271922/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE271922</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>RNA-seq in EndoCbetaH5 under high glucose, treated or not with naltrindole</name><description>In human EndoCβH5 cells, i.e. post-mitotic mature pancreatic beta cells that endogenously express OPRD1 encoding delta opioid receptor (transcripts per million [TPM] of 11 according to our RNA-seq data), a specific antagonist for delta opioid receptor (naltrindole [NTI]) significantly increased insulin secretion in high glucose condition. We performed RNA-seq in these cells to decipher signalling pathways under delta opioid receptor antagonism.</description><dates><publication>2024/07/10</publication></dates><accession>GSE271922</accession><cross_references><GSM>GSM8389150</GSM><GSM>GSM8389148</GSM><GSM>GSM8389149</GSM><GSM>GSM8389145</GSM><GSM>GSM8389146</GSM><GSM>GSM8389147</GSM><GSM>GSM8389151</GSM><GSM>GSM8389152</GSM><GPL>24676</GPL><GSE>271922</GSE><taxon>Homo sapiens</taxon><PMID>[39103322]</PMID></cross_references></HashMap>