<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE279nnn/GSE279042/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Mus musculus</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE279042</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Reprogramming of epidermal keratinocytes by PITX1 transforms the cutaneous cellular landscape and promotes wound healing [CUT&amp;Tag]</name><description>Skin injuries heal slowly, compared to oral epithelial tissues, and often do not regenerate lost adnexa. Using a murine mouse model expressing the oral epithelial transcription factor PITX1 in the skin, we performed CUT&amp;Tag-seq analysis in healthy skin to delinate PITX1 and histone occupancies in the skin.</description><dates><publication>2026/04/27</publication></dates><accession>GSE279042</accession><cross_references><GSM>GSM8560031</GSM><GSM>GSM8560053</GSM><GSM>GSM8560054</GSM><GSM>GSM8560032</GSM><GSM>GSM8560051</GSM><GSM>GSM8560052</GSM><GSM>GSM8560030</GSM><GSM>GSM8560050</GSM><GSM>GSM8560039</GSM><GSM>GSM8560037</GSM><GSM>GSM8560038</GSM><GSM>GSM8560035</GSM><GSM>GSM8560036</GSM><GSM>GSM8560055</GSM><GSM>GSM8560033</GSM><GSM>GSM8560034</GSM><GSM>GSM8560042</GSM><GSM>GSM8560043</GSM><GSM>GSM8560040</GSM><GSM>GSM8560041</GSM><GSM>GSM8560028</GSM><GSM>GSM8560029</GSM><GSM>GSM8560048</GSM><GSM>GSM8560026</GSM><GSM>GSM8560049</GSM><GSM>GSM8560027</GSM><GSM>GSM8560046</GSM><GSM>GSM8560047</GSM><GSM>GSM8560044</GSM><GSM>GSM8560045</GSM><GPL>24247</GPL><GSE>279042</GSE><taxon>Mus musculus</taxon><PMID>[39480496]</PMID></cross_references></HashMap>