<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE283nnn/GSE283630/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE283630</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Patients who received abatacept for graft versus host disease prophylaxis have lower peri-engraftment NETosis and less endothelial injury</name><description>Abatacept decreases risk of aGVHD in adult and pediatric patient populations, but its effect on other clinical outcomes of HSCT has not been well studied. Abatacept’s effect in reducing GVHD has been linked to T-cell dependent mechanisms, but it’s effect on neutrophils and completement activation is largely unknown. In this study we analyzed the effect of abatacept prophylaxis in clinical outcomes of HSCT and explored other potential mechanisms.</description><dates><publication>2026/06/30</publication></dates><accession>GSE283630</accession><cross_references><GSM>GSM8668227</GSM><GSM>GSM8668228</GSM><GSM>GSM8668225</GSM><GSM>GSM8668226</GSM><GSM>GSM8668223</GSM><GSM>GSM8668224</GSM><GPL>21697</GPL><GSE>283630</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>