{"database":"GEO","file_versions":[{"headers":{"Content-Type":["application/json"]},"body":{"files":{"Other":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE289nnn/GSE289237/"]},"type":"primary"},"statusCode":"OK","statusCodeValue":200}],"scores":null,"additional":{"omics_type":["Transcriptomics"],"species":["Homo sapiens"],"gds_type":["Expression profiling by high throughput sequencing"],"full_dataset_link":["https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE289237"],"repository":["GEO"],"entry_type":["GSE"],"additional_accession":[]},"is_claimable":false,"name":"Effect of overexpression of HMGA2 in human chondrosarcoma SW1353 cell line overexpression and control cells.","description":"Chondrosarcoma (CHS) is a malignant bone tumour resistant to adjuvant treatment, with high-grade patients suffering an unfavorable prognosis. The high mobility group A2 (HMGA2) protein is a non‑histone architectural transcription factor influencing a variety of biological processes, including the cell cycle process, DNA damage repair process, apoptosis, senescence and epithelial‑mesenchymal transition. We here aimed to investigate the role of HMGA2 in CHS.","dates":{"publication":"2026/05/27"},"accession":"GSE289237","cross_references":{"GSM":["GSM8787509","GSM8787508","GSM8787507","GSM8787506","GSM8787511","GSM8787510"],"GPL":["34284"],"GSE":["289237"],"taxon":["Homo sapiens"],"PMID":["[41964890]"]}}