<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE292nnn/GSE292301/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Mus musculus</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE292301</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>The impact of BSCI exposure on labour in pregnant mice after IL1A induction</name><description>To examine changes in chromatin accessibility involved in BSCI-mediated prevention of IL-1α-induced labour in pregnant mice, we conducted ATAC-seq on mouse myometrial tissues from four groups: Vehicle, IL-1α, BSCI and combined IL-1α with BSCI. Specifically, IL-1α is a method of labour induction, while administration of BSCI prior to and after administration of IL-1α prevents labour onset in mice. These data enable dissection of cis-regulatory elements involved in labour onset in response to IL-1α labour induction, as well as understanding of how these regions are modified in response to BSCI to prevent labour induction.</description><dates><publication>2026/06/30</publication></dates><accession>GSE292301</accession><cross_references><GSM>GSM8855790</GSM><GSM>GSM8855791</GSM><GSM>GSM8855792</GSM><GSM>GSM8855793</GSM><GSM>GSM8855794</GSM><GSM>GSM8855784</GSM><GSM>GSM8855795</GSM><GSM>GSM8855785</GSM><GSM>GSM8855786</GSM><GSM>GSM8855787</GSM><GSM>GSM8855788</GSM><GSM>GSM8855789</GSM><GPL>34328</GPL><GSE>292301</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>