<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE292nnn/GSE292556/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE292556</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Effect of hepatic FPN ablation on the liver transcriptome in mice</name><description>Iron overload has emerged as a key risk factor for MASLD and metabolic disorders. We explored the effects of hepatocyte-specific knockout of ferroportin (FPN), the only known iron exporter, on the pathogenesis of MASLD and the associated metabolic dysfunction. To investigate the molecular basis, we carried out RNA-sequencing analysis using liver samples from littermate control and hepatocyte-specific Fpn KO mice.</description><dates><publication>2026/04/14</publication></dates><accession>GSE292556</accession><cross_references><GSM>GSM8861399</GSM><GSM>GSM8861398</GSM><GSM>GSM8861401</GSM><GSM>GSM8861400</GSM><GSM>GSM8861403</GSM><GSM>GSM8861402</GSM><GPL>24247</GPL><GSE>292556</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>