<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE294nnn/GSE294892/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Rattus norvegicus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE294892</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Impaired taurine transport contributes to the development of bronchopulmonary dysplasia</name><description>Sprague-Dawley rat pups were used to study the effect of taurine supplementation on hyperoxia-exposed lungs. The study, which was conducted with meticulous attention to detail, found significant effects of taurine supplementation. Rat pups were randomly assigned to room air (NOX) and >90% oxygen (HOX) from postnatal day 1 (P1) to P10. Half of the pups of each group received a daily taurine (100 mg/kg) intraperitoneal injection or an equal volume of PBS (two by two design). The four groups were NOX-PBS, NOX-taurine, HOX-PBS, and HOX-taurine. At P10, a comprehensive range of methodologies was employed, including morphometrics, histology, metabolomics, and immunoblots. Two pups, one male and one female, were randomly selected for single-cell RNA sequencing at P10</description><dates><publication>2026/04/15</publication></dates><accession>GSE294892</accession><cross_references><GSM>GSM8927784</GSM><GSM>GSM8927781</GSM><GSM>GSM8927782</GSM><GSM>GSM8927783</GSM><GPL>20084</GPL><GSE>294892</GSE><taxon>Rattus norvegicus</taxon></cross_references></HashMap>