<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE299nnn/GSE299706/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE299706</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptome regulation in T-cells and monocytes in patients with Common Variable Immunodeficiency</name><description>A large subgroup of Common Variable Immunodeficiency (CVID) patients has autoimmune and inflammatory complications, associated with T cell and monocyte pathology, but the molecular mechanism for this CVID-subgroup is still elusive. To identify novel molecular pathways impacted in CVID T cells and monocytes, we examined the transcriptomic profiles in these cells from healthy control and the CVID patients in the subgroup with inflammatory complications.</description><dates><publication>2026/07/15</publication></dates><accession>GSE299706</accession><cross_references><GSM>GSM9044515</GSM><GSM>GSM9044525</GSM><GSM>GSM9044514</GSM><GSM>GSM9044517</GSM><GSM>GSM9044506</GSM><GSM>GSM9044516</GSM><GSM>GSM9044519</GSM><GSM>GSM9044508</GSM><GSM>GSM9044507</GSM><GSM>GSM9044518</GSM><GSM>GSM9044509</GSM><GSM>GSM9044520</GSM><GSM>GSM9044522</GSM><GSM>GSM9044511</GSM><GSM>GSM9044510</GSM><GSM>GSM9044521</GSM><GSM>GSM9044524</GSM><GSM>GSM9044513</GSM><GSM>GSM9044512</GSM><GSM>GSM9044523</GSM><GPL>18573</GPL><GSE>299706</GSE><taxon>Homo sapiens</taxon><PMID>[41991796]</PMID></cross_references></HashMap>