<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE300nnn/GSE300266/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE300266</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Short-Fiber Scaffold PGCL Stop Overgrowth for Endometrial Hyperplasia</name><description>Endometrial hyperplasia (EH), a precursor to endometrial carcinoma, poses a major threat to women’s health. Current treatments are hindered by the poor adaptability of rigid levonorgestrel (LNG)-releasing intrauterine systems. To overcome this, By physically loading LNG into the base PGC scaffold, we constructed a drug-loaded PGCL scaffold with improved therapeutic function. To elucidate the underlying molecular mechanisms of PGCL scaffold therapy, we performed high-throughput RNA sequencing on Ishikawa cells treated with PGC and PGCL scaffolds, respectively.</description><dates><publication>2026/06/30</publication></dates><accession>GSE300266</accession><cross_references><GSM>GSM9056730</GSM><GSM>GSM9056731</GSM><GSM>GSM9056732</GSM><GSM>GSM9056733</GSM><GSM>GSM9056734</GSM><GSM>GSM9056735</GSM><GPL>24676</GPL><GSE>300266</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>