<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE304nnn/GSE304381/</Other></files><type>primary</type></body><statusCodeValue>200</statusCodeValue><statusCode>OK</statusCode></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE304381</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Potential mechanisms of HOXA5 in AML by RNA-seq analysis</name><description>To explore the potential mechanisms of HOXA5 on AML, RNA-seq analysis was employed to evaluate the pattern of gene expressions in KG-1 cells with HOXA5 knockdown. Compared to the siNC group, the siHOXA5-1 group exhibited 757 differentially expressed genes (DEGs), of which 377 were upregulated and 380 were downregulated . The siHOXA5-2 group exhibited 801 DEGs, of which 387 were upregulated and 414 were downregulated. Then, we identified the DEGs after both siHOXA5-1 and siHOXA5-2 treatments, and screened out 573 DEGS strictly regulated by HOXA5.</description><dates><publication>2026/08/31</publication></dates><accession>GSE304381</accession><cross_references><GSM>GSM9149708</GSM><GSM>GSM9149707</GSM><GSM>GSM9149709</GSM><GSM>GSM9149711</GSM><GSM>GSM9149710</GSM><GSM>GSM9149704</GSM><GSM>GSM9149703</GSM><GSM>GSM9149706</GSM><GSM>GSM9149705</GSM><GPL>34284</GPL><GSE>304381</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>