{"database":"GEO","file_versions":[{"headers":{"Content-Type":["application/json"]},"body":{"files":{"Other":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE305nnn/GSE305315/"]},"type":"primary"},"statusCode":"OK","statusCodeValue":200}],"scores":null,"additional":{"omics_type":["Transcriptomics"],"species":["Homo sapiens"],"gds_type":["Expression profiling by high throughput sequencing"],"full_dataset_link":["https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE305315"],"repository":["GEO"],"entry_type":["GSE"],"additional_accession":[]},"is_claimable":false,"name":"Transcriptomic profiling of blood neutrophils from preterm infants with bronchopulmonary dysplasia","description":"Bronchopulmonary dysplasia (BPD) is a major complication of preterm birth, yet its immune mechanisms remains poorly understood. To explore the role of neutrophils in the pathogenesis of BPD, a prospective study was conducted in the Neonatal Intensive Care Unit of Guangzhou Women and Children’s Medical Center from June to September 2024. Inclusion criteria were: (1) preterm birth at <32 weeks of gestational age, and (2) admission within 6 hours after birth. Peripheral blood samples were collected on day 14 after birth. Infants with major congenital anomalies or acute infections were excluded. A total of 22 preterm infants were enrolled, of whom 12 developed BPD. Neutrophils were isolated from peripheral blood, and bulk RNA sequencing was performed.","dates":{"publication":"2026/08/13"},"accession":"GSE305315","cross_references":{"GSM":["GSM9168634","GSM9168645","GSM9168646","GSM9168635","GSM9168636","GSM9168647","GSM9168648","GSM9168637","GSM9168630","GSM9168641","GSM9168631","GSM9168642","GSM9168643","GSM9168632","GSM9168633","GSM9168644","GSM9168640","GSM9168627","GSM9168638","GSM9168628","GSM9168639","GSM9168629"],"GPL":["34284"],"GSE":["305315"],"taxon":["Homo sapiens"]}}