<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE305nnn/GSE305520/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE305520</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>The Mechanism by Which HIF-2α Regulates Adipogenic Differentiation of BMSCs</name><description>The objective of this study was to investigate whether GLP-1 regulates the adipogenic differentiation of bone marrow mesenchymal stem cells (BMSCs) and to elucidate the underlying molecular mechanism. Specifically, we aimed to determine if GLP-1 suppresses adipogenesis through modulation of the PI3K-AKT and HIF signaling pathways, and to verify the dependence of this regulatory effect on HIF-2α</description><dates><publication>2026/08/19</publication></dates><accession>GSE305520</accession><cross_references><GSM>GSM9178587</GSM><GSM>GSM9178585</GSM><GSM>GSM9178586</GSM><GSM>GSM9178579</GSM><GSM>GSM9178580</GSM><GSM>GSM9178583</GSM><GSM>GSM9178584</GSM><GSM>GSM9178581</GSM><GSM>GSM9178582</GSM><GPL>24247</GPL><GSE>305520</GSE><taxon>Mus musculus</taxon><PMID>[42425087]</PMID></cross_references></HashMap>