<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE307nnn/GSE307491/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by array</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE307491</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>TGF-alpha/EGFR signalling mediates retinoic acid-induced lung repair</name><description>Retinoic acid (RA) signalling is an important regulator of lung development and repair. To investigate the molecular mechanisms underlying RA-mediated responses in lung endothelial cells, we profiled the gene expression of human pulmonary microvascular endothelial cells (HPMECs) treated with RA or ethanol (EtOH) vehicle control. Whole-genome expression analysis using Affymetrix Human U133 Plus 2.0 GeneChips identified transcriptional changes associated with angiogenesis, wound healing, and signalling pathways relevant to alveolar repair.</description><dates><publication>2026/07/02</publication></dates><accession>GSE307491</accession><cross_references><GSM>GSM9225532</GSM><GSM>GSM9225510</GSM><GSM>GSM9225521</GSM><GSM>GSM9225520</GSM><GSM>GSM9225531</GSM><GSM>GSM9225530</GSM><GSM>GSM9225518</GSM><GSM>GSM9225529</GSM><GSM>GSM9225517</GSM><GSM>GSM9225528</GSM><GSM>GSM9225527</GSM><GSM>GSM9225516</GSM><GSM>GSM9225515</GSM><GSM>GSM9225526</GSM><GSM>GSM9225514</GSM><GSM>GSM9225525</GSM><GSM>GSM9225513</GSM><GSM>GSM9225524</GSM><GSM>GSM9225523</GSM><GSM>GSM9225512</GSM><GSM>GSM9225511</GSM><GSM>GSM9225533</GSM><GSM>GSM9225522</GSM><GSM>GSM9225519</GSM><GPL>570</GPL><GSE>307491</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>