<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE311nnn/GSE311567/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE311567</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic adaptations of human muscle in patients with polyarteritis nodosa and muscle denervation</name><description>The molecular mechanisms that govern muscle atrophy (as seen in disuse or aging) remain incompletely defined. Through RNA-seq of human muscle, we confirm observations from mice that extensive remodeling of the neuromuscular junction (NMJ) occurs in human denervated muscle.</description><dates><publication>2026/04/23</publication></dates><accession>GSE311567</accession><cross_references><GSM>GSM9327162</GSM><GSM>GSM9327161</GSM><GSM>GSM9327153</GSM><GSM>GSM9327164</GSM><GSM>GSM9327163</GSM><GSM>GSM9327160</GSM><GSM>GSM9327159</GSM><GSM>GSM9327158</GSM><GSM>GSM9327155</GSM><GSM>GSM9327165</GSM><GSM>GSM9327154</GSM><GSM>GSM9327157</GSM><GSM>GSM9327156</GSM><GPL>24676</GPL><GPL>21290</GPL><GSE>311567</GSE><taxon>Homo sapiens</taxon><PMID>[42019489]</PMID></cross_references></HashMap>