<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE312nnn/GSE312124/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE312124</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>MAPK- driven glioma progression and reprogramming of the tumor-associated immune response</name><description>Glioma evolution is associated with disease progression and therapeutic resistance. We recently identified a group of MAPK-driven glioma with CDKN2A homozygous deletion that are low-grade and immune enriched. However, Over time, these tumors can progress to aggressive, higher-grade tumors. While low-grade tumors may be amenable to immunotherapeutic strategies, it is unknown how the tumor and its microenvironment evolve.</description><dates><publication>2026/07/16</publication></dates><accession>GSE312124</accession><cross_references><GSM>GSM9338107</GSM><GSM>GSM9338108</GSM><GSM>GSM9338103</GSM><GSM>GSM9338104</GSM><GSM>GSM9338105</GSM><GSM>GSM9338106</GSM><GSM>GSM9338099</GSM><GSM>GSM9338100</GSM><GSM>GSM9338101</GSM><GSM>GSM9338102</GSM><GPL>24676</GPL><GSE>312124</GSE><taxon>Homo sapiens</taxon><PMID>[42377256]</PMID></cross_references></HashMap>