<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE312nnn/GSE312667/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Sus scrofa</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE312667</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Analysis of host gene differential expression after PAM cells were infected with ΔD250R ASFV</name><description>PAMs infected with either WT or ΔD250R ASFV revealed widespread transcriptional upregulation in ΔD250R-infected cells compared with WT-infected cells.The upregulated genes showed significant enrichment of innate immune signaling pathways, with four of the top five pathways corresponding to innate immune activation, including the cytosolic DNA-sensing, NOD-like receptor, RIG-I-like receptor, and Toll-like receptor signaling pathways.</description><dates><publication>2026/07/22</publication></dates><accession>GSE312667</accession><cross_references><GSM>GSM9351182</GSM><GSM>GSM9351174</GSM><GSM>GSM9351181</GSM><GSM>GSM9351180</GSM><GSM>GSM9351179</GSM><GSM>GSM9351176</GSM><GSM>GSM9351175</GSM><GSM>GSM9351178</GSM><GSM>GSM9351177</GSM><GPL>26351</GPL><GSE>312667</GSE><taxon>Sus scrofa</taxon></cross_references></HashMap>