<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE313nnn/GSE313813/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE313813</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Monocyte Chemokines Enhance Atherosclerotic Plaque Necrosis After Bacterial Kidney Infection</name><description>Urinary tract infections (UTI) are frequent and highly inflammatory. Using a pyelonephritis mouse model, we investigated the impact of pyelonephritis on the development of atherosclerosis. Bulk RNA sequencing analysis of kidneys and aortas harvested four weeks post-infection was performed to identify key molecular markers and delineate differentially regulated inflammatory pathways in both organs. Our results show an upregulation of inflammatory mediators in kidney and aorta, providing a possible mechanistic link between pyelonephritis and atherosclerosis.</description><dates><publication>2026/04/04</publication></dates><accession>GSE313813</accession><cross_references><GSM>GSM9376001</GSM><GSM>GSM9376012</GSM><GSM>GSM9376011</GSM><GSM>GSM9376000</GSM><GSM>GSM9376010</GSM><GSM>GSM9376005</GSM><GSM>GSM9375994</GSM><GSM>GSM9375993</GSM><GSM>GSM9376004</GSM><GSM>GSM9376003</GSM><GSM>GSM9376002</GSM><GSM>GSM9375998</GSM><GSM>GSM9376009</GSM><GSM>GSM9376008</GSM><GSM>GSM9375997</GSM><GSM>GSM9375996</GSM><GSM>GSM9376007</GSM><GSM>GSM9376006</GSM><GSM>GSM9375995</GSM><GSM>GSM9375999</GSM><GPL>24247</GPL><GSE>313813</GSE><taxon>Mus musculus</taxon><PMID>[41930659]</PMID></cross_references></HashMap>