GEOapplication/xmlftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE317nnn/GSE317003/primaryOK200GenomicsMus musculusGenome binding/occupancy profiling by high throughput sequencinghttps://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE317003GEOGSEfalseEpigenetic and transcriptional alterations in Kmt2c/Kmt2d-deficient gastric cancer [ChIP-Seq]KMT2C and KMT2D are two of the most frequently mutated genes in gastric adenocarcinoma, yet their function in cancer initiation remains poorly understood. In this study, we developed mouse models to investigate the molecular mechanism of Kmt2c/d loss in gastric tumorigenesis.2026/04/28GSE317003GSM9463695GSM9463684GSM9463694GSM9463686GSM9463685GSM9463688GSM9463687GSM9463689GSM9463691GSM9463690GSM9463693GSM946369224247317003Mus musculus