<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE317nnn/GSE317299/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE317299</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Distinct Immune Signatures of Steroid Responsiveness in Childhood Nephrotic Syndrome</name><description>Idiopathic nephrotic syndrome (INS) is the most common primary glomerular disorder in children, yet the immunopathogenesis underlying steroid responsiveness remains incompletely understood. Here, we applied single-cell RNA sequencing to peripheral blood mononuclear cells obtained from three treatment-naïve children with steroid-sensitive nephrotic syndrome (SSNS) and one child with secondary steroid-resistant nephrotic syndrome (SRNS) at first relapse, all prior to immunosuppressive therapy.</description><dates><publication>2026/07/01</publication></dates><accession>GSE317299</accession><cross_references><GSM>GSM9468896</GSM><GPL>24676</GPL><GSE>317299</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>