<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE318nnn/GSE318288/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Salmonella enterica subsp. enterica serovar Typhimurium</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE318288</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Comparative RNA seq analysis of Salmonella enterica serovar Typhimurium and a pan methionine sulfoxide reductase gene deletion (Δ5msr mutant) strain</name><description>Salmonella enterica serovar Typhimurium (S. Typhimurium) is a major foodborne pathogen which suffers but survives oxidative stress. Methionine sulfoxide reductases (Msrs) are important antioxidant enzymes that protect cells from oxidative damage by reducing oxidized methionine residues (free) or bound in proteins, back to methionine. To examine the global transcriptional consequences of deleting all five known msr genes, RNA sequencing was performed on S. Typhimurium and Δ5msr mutant strains. This dataset provides a comprehensive transcriptomic resource for investigating the impact of msrs deletion on oxidative stress–associated regulatory pathways and cellular processes in S. Typhimurium.</description><dates><publication>2026/02/05</publication></dates><accession>GSE318288</accession><cross_references><GSM>GSM9491798</GSM><GSM>GSM9491799</GSM><GSM>GSM9491796</GSM><GSM>GSM9491797</GSM><GSM>GSM9491794</GSM><GSM>GSM9491795</GSM><GPL>30218</GPL><GSE>318288</GSE><taxon>Salmonella enterica subsp. enterica serovar Typhimurium</taxon></cross_references></HashMap>