{"database":"GEO","file_versions":[{"headers":{"Content-Type":["application/json"]},"body":{"files":{"Other":["ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE318nnn/GSE318682/"]},"type":"primary"},"statusCode":"OK","statusCodeValue":200}],"scores":null,"additional":{"omics_type":["Genomics"],"species":["Homo sapiens"],"gds_type":["Genome binding/occupancy profiling by high throughput sequencing"],"full_dataset_link":["https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE318682"],"repository":["GEO"],"entry_type":["GSE"],"additional_accession":[]},"is_claimable":false,"name":"The FGF4–integrin β1 axis orchestrates diabetic wound regeneration by restoring directional collective motility [ChIP-Seq]","description":"This dataset was generated to investigate the mechanism by which Fibroblast Growth Factor 4 (FGF4) regulates the binding of C-FOS to the ITGB1 promoter. ChIP sequencing was performed on high glucose-induced HacaT cells, which were divided into low glucose, high glucose, and high glucose plus FGF4 treatment groups.","dates":{"publication":"2026/05/01"},"accession":"GSE318682","cross_references":{"GSM":["GSM9500709","GSM9500708","GSM9500707"],"GPL":["34284"],"GSE":["318682"],"taxon":["Homo sapiens"]}}