<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE325nnn/GSE325101/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE325101</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Ginsenoside Rg3 attenuates allergic asthma in mice via transcriptomics</name><description>ObjectivesThis study aims to investigate the molecular mechanism by which ginsenoside Rg3 attenuates allergic asthma. Methods: This study established an ovalbumin (OVA)-induced mouse model of allergic asthma to explore the anti-allergic effects and potential molecular mechanisms of ginsenoside Rg3. The therapeutic effects of Rg3 were systematically evaluated via monitoring of general physical conditions, lung histopathological examination, inflammatory cell infiltration analysis in bronchoalveolar lavage fluid, and inflammatory cytokine detection. Furthermore, transcriptomic analyses were conducted to screen differentially expressed genes.</description><dates><publication>2026/04/08</publication></dates><accession>GSE325101</accession><cross_references><GSM>GSM9595882</GSM><GSM>GSM9595893</GSM><GSM>GSM9595892</GSM><GSM>GSM9595881</GSM><GSM>GSM9595884</GSM><GSM>GSM9595894</GSM><GSM>GSM9595883</GSM><GSM>GSM9595886</GSM><GSM>GSM9595885</GSM><GSM>GSM9595888</GSM><GSM>GSM9595887</GSM><GSM>GSM9595891</GSM><GSM>GSM9595880</GSM><GSM>GSM9595890</GSM><GSM>GSM9595889</GSM><GPL>24247</GPL><GSE>325101</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>