<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE326nnn/GSE326402/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE326402</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Variants in PATL1 can cause intellectual disability in humans revealed by modeling in Drosophila and C. Elegans</name><description>P-bodies are dynamic, membrane-less organelles that function in mRNA metabolism that are involved in mRNA decay, storage, and translational repression. Here we introduce a new variant of the P-body protein encoding gene; PATL1, identified in an ID patient.</description><dates><publication>2026/04/10</publication></dates><accession>GSE326402</accession><cross_references><GSM>GSM9630464</GSM><GSM>GSM9630463</GSM><GSM>GSM9630468</GSM><GSM>GSM9630467</GSM><GSM>GSM9630466</GSM><GSM>GSM9630465</GSM><GPL>29480</GPL><GSE>326402</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>