<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE328nnn/GSE328018/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE328018</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Choroid plexus inflammation in bipolar disorder</name><description>Postmortem human choroid plexus tissue from individuals with bipolar disorder and matched controls was analyzed using cytokine protein arrays and bulk RNA sequencing to characterize immune and barrier-related alterations. The study identifies increased pro-inflammatory signaling and reduced trophic/supportive pathways, implicating the choroid plexus as a neuroimmune interface in bipolar disorder.</description><dates><publication>2026/06/18</publication></dates><accession>GSE328018</accession><cross_references><GSM>GSM9670419</GSM><GSM>GSM9670429</GSM><GSM>GSM9670418</GSM><GSM>GSM9670428</GSM><GSM>GSM9670417</GSM><GSM>GSM9670427</GSM><GSM>GSM9670416</GSM><GSM>GSM9670426</GSM><GSM>GSM9670415</GSM><GSM>GSM9670425</GSM><GSM>GSM9670414</GSM><GSM>GSM9670424</GSM><GSM>GSM9670413</GSM><GSM>GSM9670423</GSM><GSM>GSM9670412</GSM><GSM>GSM9670422</GSM><GSM>GSM9670411</GSM><GSM>GSM9670432</GSM><GSM>GSM9670421</GSM><GSM>GSM9670410</GSM><GSM>GSM9670431</GSM><GSM>GSM9670420</GSM><GSM>GSM9670430</GSM><GPL>34281</GPL><GSE>328018</GSE><taxon>Homo sapiens</taxon><PMID>[41980683]</PMID></cross_references></HashMap>