<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE328nnn/GSE328236/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Mus musculus</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE328236</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic profiling of Carfilzomib (CFZ) treatment in HSV-1 infected BV2 microglial cells</name><description>This study investigates the molecular mechanisms underlying the interaction between Carfilzomib (CFZ) and Herpes Simplex Virus type 1 (HSV-1) infection in microglial cells. Using the BV2 microglial cell line, we performed RNA sequencing to dissect the transcriptional responses under different conditions. Cells were either infected with HSV-1 (MOI=1) for 24 hours or left uninfected, and subsequently treated with 30 nM Carfilzomib (CFZ) or the vehicle control (DMSO) 6 hours post infection.</description><dates><publication>2026/04/21</publication></dates><accession>GSE328236</accession><cross_references><GSM>GSM9675639</GSM><GSM>GSM9675641</GSM><GSM>GSM9675640</GSM><GSM>GSM9675643</GSM><GSM>GSM9675642</GSM><GSM>GSM9675645</GSM><GSM>GSM9675644</GSM><GSM>GSM9675636</GSM><GSM>GSM9675635</GSM><GSM>GSM9675646</GSM><GSM>GSM9675638</GSM><GSM>GSM9675637</GSM><GPL>17021</GPL><GSE>328236</GSE><taxon>Mus musculus</taxon></cross_references></HashMap>