<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE328nnn/GSE328335/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Genomics</omics_type><species>Homo sapiens</species><gds_type>Genome binding/occupancy profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE328335</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic and epigenomic analysis of WSTF knockdown in colorectal cancer cells [ChIP-Seq]</name><description>This study investigates the role of Williams syndrome transcription factor (WSTF/BAZ1B) in colorectal cancer (CRC) progression through multi-omics approaches. WSTF was knocked down in SW620 colorectal cancer cells using siRNA to assess its impact on gene expression and metabolism. Additionally, WSTF overexpression was performed followed by ChIP-seq to identify genome-wide binding sites and direct transcriptional targets. The study aims to elucidate WSTF-mediated regulatory mechanisms and identify potential downstream effectors including GLYCTK in CRC.</description><dates><publication>2026/04/22</publication></dates><accession>GSE328335</accession><cross_references><GSM>GSM9679486</GSM><GSM>GSM9679485</GSM><GPL>24676</GPL><GSE>328335</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>