<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE328nnn/GSE328387/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE328387</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>LSD1 Attenuates Ferroptosis Sensitivity in Colorectal Cancer Cells via a Demethylase-Independent Manner: An Innovative Antitumor Strategy via Targeted Degradation of LSD1</name><description>To investigate the global gene expression profiles and identify differentially expressed genes, key signaling pathways, and potential molecular mechanisms underlying compound treatment in human cancer cells, RNA-seq was performed to analyze transcriptomic changes, aiming to provide comprehensive insights into the transcriptome regulation and discover novel biomarkers or therapeutic targets for CRC.</description><dates><publication>2026/04/22</publication></dates><accession>GSE328387</accession><cross_references><GSM>GSM9681773</GSM><GSM>GSM9681765</GSM><GSM>GSM9681770</GSM><GSM>GSM9681772</GSM><GSM>GSM9681771</GSM><GSM>GSM9681769</GSM><GPL>24676</GPL><GSE>328387</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>