<HashMap><database>GEO</database><file_versions><headers><Content-Type>application/xml</Content-Type></headers><body><files><Other>ftp://ftp.ncbi.nlm.nih.gov/geo/series/GSE329nnn/GSE329766/</Other></files><type>primary</type></body><statusCode>OK</statusCode><statusCodeValue>200</statusCodeValue></file_versions><scores/><additional><omics_type>Transcriptomics</omics_type><species>Homo sapiens</species><gds_type>Expression profiling by high throughput sequencing</gds_type><full_dataset_link>https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE329766</full_dataset_link><repository>GEO</repository><entry_type>GSE</entry_type></additional><is_claimable>false</is_claimable><name>Transcriptomic profiling of BAZ1A-AS1 and BAZ1A knockdown in human vascular smooth muscle cells and rhabdomyoblasts</name><description>BAZ1A-AS1 is a novel human-specific long non-coding RNA upregulated during neointima proliferation in human saphenous veins. To elucidate the transcriptional programs regulated by BAZ1A-AS1 and its cis-regulatory gene BAZ1A, we performed bulk RNA-seq on human saphenous vein smooth muscle cells (HSVSMCs) with BAZ1A-AS1 knockdown (LNA gapmeR ASO) and rhabdomyoblast (RD) cells with BAZ1A knockdown (siRNA) under UV exposure. Differential expression analysis revealed convergent suppression of proliferative, inflammatory, and stress-response transcriptional programs across both knockdowns, supporting a shared regulatory axis.</description><dates><publication>2026/07/01</publication></dates><accession>GSE329766</accession><cross_references><GSM>GSM9710849</GSM><GSM>GSM9710859</GSM><GSM>GSM9710858</GSM><GSM>GSM9710857</GSM><GSM>GSM9710856</GSM><GSM>GSM9710855</GSM><GSM>GSM9710854</GSM><GSM>GSM9710853</GSM><GSM>GSM9710852</GSM><GSM>GSM9710851</GSM><GSM>GSM9710850</GSM><GSM>GSM9710860</GSM><GPL>16791</GPL><GSE>329766</GSE><taxon>Homo sapiens</taxon></cross_references></HashMap>